Volume 13 | Issue 2
Volume 13 | Issue 2
Volume 13 | Issue 2
Volume 13 | Issue 1
Volume 13 | Issue 1
People with decompensated cirrhosis frequently experience acute renal damage, which is associated with a poor prognosis. The most common and readily recognizable cause of cirrhosis is hepatorenal syndrome type 1 (HRS-1), also known as hepatorenal syndrome-acute kidney damage. Acute kidney damage with direct links to chronic liver illness includes abdominal compartment syndrome, cardiorenal processes associated with cirrhotic cardiomyopathy and portopulmonary hypertension, and cholemic nephropathy. Some glomerular nephropathies (GNs), such as viral hepatitis and Immunoglobulin A (IgA) nephropathy, can result in acute kidney injury in cirrhosis. Understanding a medical condition's underlying etiology is essential to finding the best therapy. The best form of rehydration therapy for prerenal acute kidney damage is albumin. Both acute tubular damage and acute interstitial nephritis can be effectively treated with supportive care, removal of the underlying cause, and, in the case of the latter, corticosteroids. Acute kidney injury (AKI) is a complication of cirrhosis that calls for a comprehensive strategy to diagnose and treat it.